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R.JinX.et al (1999)

effectively collected HucMSCs-EXOs which could partly reverse Soluble fms-like tyrosine kinase-1 (sFlt-1) triggered HUVECs dysfunction in vitro

[1] Chelation therapy has a long history of use in clinical toxicology [2] and remains in use for some very specific medical treatments, although it is administered under very careful medical supervision due to various inherent risks, including the mobilization of mercury and other metals through the brain and other parts of the body by the use of weak chelating agents that unbind with metals before elimination, exacerbating existing damage

Physiological and molecular mechanisms of methionine restriction

NK cells enhance the proliferation of T cells and Mtb specific CD8 + T cells, essential for anti-TB immunity (171)

Inhibition of interleukin-1 by an interleukin-1 receptor antagonist prevents graft-versus-host disease